Glycogen Storage Disease GSD

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2025年12月22日 (月) 00:53時点におけるBrandie7668 (トーク | 投稿記録)による版
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Such a scheme serves as a template for the lactate shuttling present in the mammalian CNS. An almost contemporaneous report prolonged this scheme to use to the mammalian mind, however with notable differences and additions. It was to show one of the crucial contentious papers in the field of mind vitality metabolism and the debate raging round the subject persists. The paper described experiments carried out using cultures of mouse cerebral neurones and astrocytes, and demonstrated a couple of key features, which were stoichiometrically sturdy (Pellerin and Magistretti, 1994). The background to the study was the realization that if metabolic signaling were to occur between neurones and astrocytes in the mammalian CNS, there should exist a signaling mechanism whereby the neurones sign to astrocytes their want for delivery of metabolic substrate. The signaling mechanism will need to have the following characteristics: (1) it have to be related to the frequency of activity of the neurones i.e., the release of the compound have to be related to the exercise of the neurones, (2) it must be sensed by the astrocyte i.e., there have to be mechanism(s) whereby the astrocyte senses the molecule, and (3) there should exist a system or Healthy Flow Wellness process whereby the concentration of the molecule is decreased upon cessation of exercise i.e., there must be an effective buffering system that reduces the concentration of the molecule after exercise.

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